Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. Alternatively, our capacity to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK indicates elevated FAK signaling downstream from the Pro32Pro33 mutant, as confirmed by amplified pFAK localization in 5-HT synapses. Our information https://garrys653ueq5.goabroadblog.com/profile